Early exercise training after myocardial infarction prevents contractile but not electrical remodelling or hypertrophy
CARDIOVASCULAR RESEARCH, cilt.86, sa.1, ss.72-81, 2010 (SCI-Expanded)
- Yayın Türü: Makale / Tam Makale
- Cilt numarası: 86 Sayı: 1
- Basım Tarihi: 2010
- Doi Numarası: 10.1093/cvr/cvp381
- Dergi Adı: CARDIOVASCULAR RESEARCH
- Derginin Tarandığı İndeksler: Science Citation Index Expanded (SCI-EXPANDED), Scopus
- Sayfa Sayıları: ss.72-81
- Anahtar Kelimeler: Calcium, Exercise, Arrhythmias, Myocardial infarction, Potassium currents, MOUSE VENTRICULAR MYOCYTES, ACTION-POTENTIAL DURATION, HEART-FAILURE, SARCOPLASMIC-RETICULUM, CARDIAC-HYPERTROPHY, K+ CURRENTS, VOLUNTARY EXERCISE, CA2+ TRANSIENTS, RAT, MECHANISMS
- Açık Arşiv Koleksiyonu: AVESİS Açık Erişim Koleksiyonu
- Akdeniz Üniversitesi Adresli: Evet
Özet
Exercise started early after myocardial infarction (MI) improves in vivo cardiac function and myofilament responsiveness to Ca2+. We investigated whether this represents partial or complete reversal of cellular remodelling.