Early exercise training after myocardial infarction prevents contractile but not electrical remodelling or hypertrophy

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Bito V., de Waard M. C., Biesmans L., Lenaerts I., Ozdemir S., van Deel E., ...Daha Fazla

CARDIOVASCULAR RESEARCH, cilt.86, sa.1, ss.72-81, 2010 (SCI-Expanded) identifier identifier identifier

  • Yayın Türü: Makale / Tam Makale
  • Cilt numarası: 86 Sayı: 1
  • Basım Tarihi: 2010
  • Doi Numarası: 10.1093/cvr/cvp381
  • Dergi Adı: CARDIOVASCULAR RESEARCH
  • Derginin Tarandığı İndeksler: Science Citation Index Expanded (SCI-EXPANDED), Scopus
  • Sayfa Sayıları: ss.72-81
  • Anahtar Kelimeler: Calcium, Exercise, Arrhythmias, Myocardial infarction, Potassium currents, MOUSE VENTRICULAR MYOCYTES, ACTION-POTENTIAL DURATION, HEART-FAILURE, SARCOPLASMIC-RETICULUM, CARDIAC-HYPERTROPHY, K+ CURRENTS, VOLUNTARY EXERCISE, CA2+ TRANSIENTS, RAT, MECHANISMS
  • Akdeniz Üniversitesi Adresli: Evet

Özet

Exercise started early after myocardial infarction (MI) improves in vivo cardiac function and myofilament responsiveness to Ca2+. We investigated whether this represents partial or complete reversal of cellular remodelling.