Increased serum levels of apoptosis in deficit syndrome schizophrenia patients: a preliminary study

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BEYAZYÜZ M., Kufeciler T., Bulut L., Unsal C., ALBAYRAK Y., Akyol E. S., ...More

NEUROPSYCHIATRIC DISEASE AND TREATMENT, vol.12, pp.1261-1268, 2016 (SCI-Expanded)

  • Publication Type: Article / Article
  • Volume: 12
  • Publication Date: 2016
  • Doi Number: 10.2147/ndt.s106993
  • Journal Name: NEUROPSYCHIATRIC DISEASE AND TREATMENT
  • Journal Indexes: Science Citation Index Expanded (SCI-EXPANDED), Scopus
  • Page Numbers: pp.1261-1268
  • Keywords: apoptosis, etiology, deficit, psychosis, NEUROTROPHIC FACTOR, OXIDATIVE STRESS, MECHANISMS, NEURODEGENERATION, PATHOPHYSIOLOGY, ANTIOXIDANT, EXPRESSION, SCHEDULE, CANCER, CORTEX
  • Open Archive Collection: AVESIS Open Access Collection
  • Akdeniz University Affiliated: Yes

Abstract

Background: Schizophrenia is a chronic and debilitating disorder, the etiology of which remains unclear. Apoptosis is a programmed cell death mechanism that might be implicated in neuropsychiatric disorders, including schizophrenia. In this study, we aimed to compare the serum levels of apoptosis among deficit schizophrenia (DS) syndrome patients, nondeficit schizophrenia (NDS) patients, and healthy controls (HCs).