Increased serum levels of apoptosis in deficit syndrome schizophrenia patients: a preliminary study

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BEYAZYÜZ M., Kufeciler T., Bulut L., Unsal C., ALBAYRAK Y., Akyol E. S., ...Daha Fazla

NEUROPSYCHIATRIC DISEASE AND TREATMENT, cilt.12, ss.1261-1268, 2016 (SCI-Expanded)

  • Yayın Türü: Makale / Tam Makale
  • Cilt numarası: 12
  • Basım Tarihi: 2016
  • Doi Numarası: 10.2147/ndt.s106993
  • Dergi Adı: NEUROPSYCHIATRIC DISEASE AND TREATMENT
  • Derginin Tarandığı İndeksler: Science Citation Index Expanded (SCI-EXPANDED), Scopus
  • Sayfa Sayıları: ss.1261-1268
  • Anahtar Kelimeler: apoptosis, etiology, deficit, psychosis, NEUROTROPHIC FACTOR, OXIDATIVE STRESS, MECHANISMS, NEURODEGENERATION, PATHOPHYSIOLOGY, ANTIOXIDANT, EXPRESSION, SCHEDULE, CANCER, CORTEX
  • Açık Arşiv Koleksiyonu: AVESİS Açık Erişim Koleksiyonu
  • Akdeniz Üniversitesi Adresli: Evet

Özet

Background: Schizophrenia is a chronic and debilitating disorder, the etiology of which remains unclear. Apoptosis is a programmed cell death mechanism that might be implicated in neuropsychiatric disorders, including schizophrenia. In this study, we aimed to compare the serum levels of apoptosis among deficit schizophrenia (DS) syndrome patients, nondeficit schizophrenia (NDS) patients, and healthy controls (HCs).