Triamcinolone up-regulates GLUT 1 and GLUT 3 expression in cultured human placental endothelial cells
CELL BIOCHEMISTRY AND FUNCTION, cilt.30, sa.1, ss.47-53, 2012 (SCI-Expanded)
- Yayın Türü: Makale / Tam Makale
- Cilt numarası: 30 Sayı: 1
- Basım Tarihi: 2012
- Doi Numarası: 10.1002/cbf.1817
- Dergi Adı: CELL BIOCHEMISTRY AND FUNCTION
- Derginin Tarandığı İndeksler: Science Citation Index Expanded (SCI-EXPANDED), Scopus
- Sayfa Sayıları: ss.47-53
- Anahtar Kelimeler: glucocorticoids, glucose transport, pregnancy, fetal growth, GLUCOSE-TRANSPORTER EXPRESSION, GLUCOCORTICOID-RECEPTOR, PROTEIN EXPRESSION, INSULIN-RESISTANCE, FETAL-GROWTH, DEXAMETHASONE, EXPOSURE, SYNCYTIOTROPHOBLAST, DECIDUALIZATION, IMPLANTATION
- Akdeniz Üniversitesi Adresli: Evet
Özet
The placenta is a glucocorticoid target organ, and glucocorticoids (GCs) are essential for the development and maturation of fetal organs. They are widely used for treatment of a variety of diseases during pregnancy. In various tissues, GCs have regulated by glucose transport systems; however, their effects on glucose transporters in the human placental endothelial cells (HPECs) are unknown.