The role of selenium in bevacizumab induced cardiotoxicity
BRATISLAVA MEDICAL JOURNAL-BRATISLAVSKE LEKARSKE LISTY, cilt.120, sa.2, ss.131-138, 2019 (SCI-Expanded, Scopus)
- Yayın Türü: Makale / Tam Makale
- Cilt numarası: 120 Sayı: 2
- Basım Tarihi: 2019
- Doi Numarası: 10.4149/bll_2019_021
- Dergi Adı: BRATISLAVA MEDICAL JOURNAL-BRATISLAVSKE LEKARSKE LISTY
- Derginin Tarandığı İndeksler: Science Citation Index Expanded (SCI-EXPANDED), Scopus
- Sayfa Sayıları: ss.131-138
- Anahtar Kelimeler: apoptosis, bevacizumab, cardiomyocyte, transient receptor potential vanilloid 1, selenium, MITOCHONDRIAL OXIDATIVE STRESS, ENDOTHELIAL GROWTH-FACTOR, CANCER CELLS INVOLVEMENT, CARDIAC-HYPERTROPHY, CHANNEL EXPRESSION, CALCIUM OVERLOAD, HEART-FAILURE, TRPV1, APOPTOSIS, RECEPTOR
- Açık Arşiv Koleksiyonu: AVESİS Açık Erişim Koleksiyonu
- Akdeniz Üniversitesi Adresli: Evet
Özet
OBJECTIVE: We investigated the role of selenium in bevacizumab induced cardiotoxicity and involvement of transient receptor potential vanilloid 1 (TRPV1) channels in cardiomyocytes.