The role of selenium in bevacizumab induced cardiotoxicity

Creative Commons License

Oncel C. R., Ovey I. S.

BRATISLAVA MEDICAL JOURNAL-BRATISLAVSKE LEKARSKE LISTY, cilt.120, sa.2, ss.131-138, 2019 (SCI-Expanded) identifier identifier identifier

  • Yayın Türü: Makale / Tam Makale
  • Cilt numarası: 120 Sayı: 2
  • Basım Tarihi: 2019
  • Doi Numarası: 10.4149/bll_2019_021
  • Dergi Adı: BRATISLAVA MEDICAL JOURNAL-BRATISLAVSKE LEKARSKE LISTY
  • Derginin Tarandığı İndeksler: Science Citation Index Expanded (SCI-EXPANDED), Scopus
  • Sayfa Sayıları: ss.131-138
  • Anahtar Kelimeler: apoptosis, bevacizumab, cardiomyocyte, transient receptor potential vanilloid 1, selenium, MITOCHONDRIAL OXIDATIVE STRESS, ENDOTHELIAL GROWTH-FACTOR, CANCER CELLS INVOLVEMENT, CARDIAC-HYPERTROPHY, CHANNEL EXPRESSION, CALCIUM OVERLOAD, HEART-FAILURE, TRPV1, APOPTOSIS, RECEPTOR
  • Açık Arşiv Koleksiyonu: AVESİS Açık Erişim Koleksiyonu
  • Akdeniz Üniversitesi Adresli: Evet

Özet

OBJECTIVE: We investigated the role of selenium in bevacizumab induced cardiotoxicity and involvement of transient receptor potential vanilloid 1 (TRPV1) channels in cardiomyocytes.