Interleukin-33 could play an important role in the pathogenesis of periodontitis

Koseoglu S., HATİPOĞLU M., Saglam M., Enhos S., Esen H. H.

JOURNAL OF PERIODONTAL RESEARCH, cilt.50, sa.4, ss.525-534, 2015 (SCI-Expanded) identifier identifier identifier

  • Yayın Türü: Makale / Tam Makale
  • Cilt numarası: 50 Sayı: 4
  • Basım Tarihi: 2015
  • Doi Numarası: 10.1111/jre.12235
  • Dergi Adı: JOURNAL OF PERIODONTAL RESEARCH
  • Derginin Tarandığı İndeksler: Science Citation Index Expanded (SCI-EXPANDED), Scopus
  • Sayfa Sayıları: ss.525-534
  • Anahtar Kelimeler: alveolar bone loss, interleukin-33, osteoprotegerin, periodontal disease, RANKL protein, rat, RECEPTOR ACCESSORY PROTEIN, GINGIVAL CREVICULAR FLUID, ALVEOLAR BONE LOSS, KAPPA-B LIGAND, RHEUMATOID-ARTHRITIS, CYTOKINE IL-33, T-LYMPHOCYTES, ST2 COMPRISE, TH2 CELLS, IN-VIVO
  • Akdeniz Üniversitesi Adresli: Evet

Özet

Background and ObjectiveInterleukin-33 (IL-33) controls T-helper type 2 (Th2) cytokines and the development of mast cells. This study aimed to investigate the expression of IL-33 and its association with RANKL and osteoprotegerin (OPG) in periodontal health and experimental periodontitis.